Immunodetectable levels of the inhibitory guanine nucleotide-binding regulatory proteins in failing human heart: Discordance with measurements of adenylate cyclase activity and levels of pertussis toxin substrate

This publication has 30 references indexed in Scilit:

• Localization of a “postreceptor” defect in human dilated cardiomyopathy
  The American Journal of Cardiology, 1989
• Altered expression of α-subunits of G proteins in failing human hearts
  Journal of Molecular and Cellular Cardiology, 1989
• Adenylate cyclase activity coupled to the stimulatory guanine nucleotide binding protein in patients having electrophysiologic studies and either structurally normal hearts or idiopathic myocardial disease
  The American Journal of Cardiology, 1988
• Effects of phosphorylation of inhibitory GTP‐binding protein by cyclic AMP‐dependent protein kinase on its ADP‐ribosylation by pertussis toxin, islet‐activating protein
  FEBS Letters, 1988
• Decreased stimulatory guanosine triphosphate binding protein in dogs with pressure-overload left ventricular failure.
  Journal of Clinical Investigation, 1988
• Reconstitution of the purified porcine atrial muscarinic acetylcholine receptor with purified porcine atrial inhibitory guanine nucleotide binding protein
  Biochemistry, 1987
• NAD+-mediated stimulation of adenylate cyclase in cardiac membranes
  Biochemical and Biophysical Research Communications, 1987
• Regulation of adenylate cyclase by hormones and G‐proteins
  FEBS Letters, 1987
• Protein kinase C phosphorylates the inhibitory guanine‐nucleotide‐binding regulatory component and apparently suppresses its function in hormonal inhibition of adenylate cyclase
  European Journal of Biochemistry, 1985
• Decreased Catecholamine Sensitivity and β-Adrenergic-Receptor Density in Failing Human Hearts
  New England Journal of Medicine, 1982