Bax is required for increased enterocyte apoptosis after massive small bowel resection

This publication has 18 references indexed in Scilit:

• Effect of massive small bowel resection on the Bax/Bcl-w ratio and enterocyte apoptosis
  Journal of Gastrointestinal Surgery, 2000
• p21 Promotes Ceramide-Induced Apoptosis and Antagonizes the Antideath Effect of Bcl-2 in Human Hepatocarcinoma Cells
  Experimental Cell Research, 1999
• Survival activity of Bcl-2 homologs Bcl-w and A1 only partially correlates with their ability to bind pro-apoptotic family members
  Cell Death & Differentiation, 1999
• Bax cleavage is mediated by calpain during drug-induced apoptosis
  Oncogene, 1998
• The Bcl-2 Protein Family: Arbiters of Cell Survival
  Science, 1998
• Movement of Bax from the Cytosol to Mitochondria during Apoptosis
  The Journal of cell biology, 1997
• Epidermal growth factor and human growth hormone accelerate adaptation after massive enterectomy in an additive, nutrient-dependent, and site-specific fashion
  Surgery, 1997
• Interaction of CED-4 with CED-3 and CED-9: A Molecular Framework for Cell Death
  Science, 1997
• Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programed cell death
  Cell, 1993
• Intestinal Adaptation
  New England Journal of Medicine, 1978