Chronic Δ-9-tetrahydrocannabinol treatment increases cAMP levels and cAMP-dependent protein kinase activity in some rat brain regions
- 30 June 2000
- journal article
- Published by Elsevier in Neuropharmacology
- Vol. 39 (7) , 1331-1336
- https://doi.org/10.1016/s0028-3908(99)00196-3
Abstract
No abstract availableKeywords
This publication has 20 references indexed in Scilit:
- Dual intracellular signaling pathways mediated by the human cannabinoid CB1 receptorEuropean Journal of Pharmacology, 1999
- Cannabinoid receptor down-regulation without alteration of the inhibitory effect of CP 55,940 on adenylyl cyclase in the cerebellum of CP 55,940-tolerant micePublished by Elsevier ,1999
- Behavioural and biochemical evidence for signs of abstinence in mice chronically treated with Δ‐9‐tetrahydrocannabinolBritish Journal of Pharmacology, 1998
- Concurrent Stimulation of Cannabinoid CB1 and Dopamine D2 Receptors Augments cAMP Accumulation in Striatal Neurons: Evidence for a GsLinkage to the CB1 ReceptorJournal of Neuroscience, 1997
- Inhibition of protein kinase A and cyclic AMP response element (CRE)-specific transcription factor binding by Δ9-tetrahydrocannabinol (Δ9-THC): A putative mechanism of cannabinoid-induced immune modulationBiochemical Pharmacology, 1997
- Chronic cannabinoid, CP-55,940, administration alters biotransformation in the ratEuropean Journal of Pharmacology, 1996
- Development of behavioral tolerance to Δ9-THC without alteration of cannabinoid receptor binding or mRNA levels in whole brainPharmacology Biochemistry and Behavior, 1993
- Chronic cannabinoid administration alters cannabinoid receptor binding in rat brain: a quantitative autoradiographic studyBrain Research, 1993
- Cannabinoid Receptors and Modulation of Cyclic AMP Accumulation in the Rat BrainJournal of Neurochemistry, 1990
- A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye bindingAnalytical Biochemistry, 1976