Lithium Suppresses Signaling and Induces Rapid Sequestration of β2-Adrenergic Receptors
- 19 October 2001
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 288 (1) , 151-155
- https://doi.org/10.1006/bbrc.2001.5755
Abstract
No abstract availableKeywords
This publication has 19 references indexed in Scilit:
- c-Src Tyrosine Kinase Binds the β2-Adrenergic Receptor via Phospho-Tyr-350, Phosphorylates G-protein-linked Receptor Kinase 2, and Mediates Agonist-induced Receptor DesensitizationJournal of Biological Chemistry, 2001
- Mood Stabilizers and Ion RegulationHarvard Review of Psychiatry, 2001
- The superfamily of heptahelical receptorsNature Cell Biology, 2000
- Gravin-mediated Formation of Signaling Complexes in β2-Adrenergic Receptor Desensitization and ResensitizationJournal of Biological Chemistry, 2000
- Racial variation in antidepressant treatment in a Medicaid population.The Journal of Clinical Psychiatry, 2000
- Different mechanisms of protection against apoptosis by valproate and Li+European Journal of Biochemistry, 1999
- Lithium activates the serine/threonine kinase Akt-1 and suppresses glutamate-induced inhibition of Akt-1 activity in neuronsProceedings of the National Academy of Sciences, 1999
- Physiological Regulation of G Protein-Linked SignalingPhysiological Reviews, 1999
- Indirect immunofluorescence localization of β-adrenergic receptors and G-proteins in human A431 cellsBiochemical Journal, 1989
- The Effects of Lithium on myo‐Inositol Levels in Layers of Frontal Cerebral Cortex, in Cerebellum, and in Corpus Callosum of the RatJournal of Neurochemistry, 1980