Alzheimer’s amyloid β-peptide (1–42) induces cell death in human neuroblastoma via bax/bcl-2 ratio increase: An intriguing role for methionine 35
- 31 March 2006
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 342 (1) , 206-213
- https://doi.org/10.1016/j.bbrc.2006.01.137
Abstract
No abstract availableKeywords
This publication has 70 references indexed in Scilit:
- Aβ(31–35) peptide induce apoptosis in PC 12 cells: Contrast with Aβ(25–35) peptide and examination of underlying mechanismsNeurochemistry International, 2005
- β-Amyloid-induced apoptosis is associated with cyclooxygenase-2 up-regulation via the mitogen-activated protein kinase–NF-κB signaling pathwayFree Radical Biology & Medicine, 2005
- Aβ(31–35) and Aβ(25–35) fragments of amyloid beta‐protein induce cellular death through apoptotic signals: Role of the redox state of methionine‐35FEBS Letters, 2005
- Amyloid β‐protein fragment 31–35 suppresses delayed rectifying potassium channels in membrane patches excised from hippocampal neurons in ratsSynapse, 2003
- Neurotoxic, Redox-competent Alzheimer's β-Amyloid Is Released from Lipid Membrane by Methionine OxidationJournal of Biological Chemistry, 2003
- Bcl-xS can form homodimers and heterodimers and its apoptotic activity requires localization of Bcl-xS to the mitochondria and its BH3 and loop domainsCell Death & Differentiation, 2001
- E2F1 Mediates Death of B-amyloid-treated Cortical Neurons in a Manner Independent of p53 and Dependent on Bax and Caspase 3Journal of Biological Chemistry, 2000
- Amelioration of Hippocampal Neuronal Damage After Global Ischemia by Neuronal Overexpression of BCL-2 in Transgenic MiceStroke, 1998
- Inhibitors of permeability transition interfere with the disruption of the mitochondrial transmembrane potential during apoptosisFEBS Letters, 1996
- Physical, Morphological and Functional Differences between pH 5.8 and 7.4 Aggregates of the Alzheimer's Amyloid Peptide A βJournal of Molecular Biology, 1996