Dominant-negative Mutants of Grb2 Induced Reversal of the Transformed Phenotypes Caused by the Point Mutation-activated Rat HER-2/Neu
Open Access
- 1 December 1995
- journal article
- Published by Elsevier
- Vol. 270 (51) , 30717-30724
- https://doi.org/10.1074/jbc.270.51.30717
Abstract
No abstract availableKeywords
This publication has 38 references indexed in Scilit:
- Association of Sos Ras exchange protein with Grb2 is implicated in tyrosine kinase signal transduction and transformationNature, 1993
- Guanine-nucleotide-releasing factor hSos1 binds to Grb2 and links receptor tyrosine kinases to Ras signallingNature, 1993
- The SH2 and SH3 domains of mammalian Grb2 couple the EGF receptor to the Ras activator mSos1Nature, 1993
- Oral cancer progression and c-erbB-2/neu proto-oncogene expressionCancer Letters, 1992
- Mechanistic Aspects of Signaling Through Ras in NIH 3T3 CellsScience, 1992
- A novel transforming protein (SHC) with an SH2 domain is implicated in mitogenic signal transductionCell, 1992
- Neu differentiation factor: A transmembrane glycoprotein containing an EGF domain and an immunoglobulin homology unitCell, 1992
- Direct Interaction of a Ligand for the erb B2 Oncogene Product with the EGF Receptor and p185
erb
B2
Science, 1990
- Neu receptor dimerizationNature, 1989
- Tyrosine Kinase Receptor with Extensive Homology to EGF Receptor Shares Chromosomal Location with neu OncogeneScience, 1985