Effects of cocaine, nicotine, dizocipline and alcohol on mice locomotor activity: cocaine–alcohol cross-sensitization involves upregulation of striatal dopamine transporter binding sites

This publication has 37 references indexed in Scilit:

• Open-Channel Blockers at the Human α4β2 Neuronal Nicotinic Acetylcholine Receptor
  Molecular Pharmacology, 1998
• Signalled versus unsignalled intravenous amphetamine: large differences in the acute psychomotor response and sensitization
  Brain Research, 1996
• Conditioned activation induced by ethanol: Role in sensitization and conditioned place preference
  Pharmacology Biochemistry and Behavior, 1992
• Genetic differences in the rewarding and activating effects of morphine and ethanol
  Psychopharmacology, 1992
• Effects of nicotinic agonists on the NMDA receptor
  Brain Research, 1991
• Unaltered [3H]GBR-12935 binding after chronic treatment with dopamine active drugs
  Psychopharmacology, 1990
• Nicotine reduces the binding of [3H]MK-801 to brain membranes, but not via the stimulation of high-affinity nicotinic receptors
  Brain Research, 1990
• The NMDA antagonist MK-801 causes marked locomotor stimulation in monoamine-depleted mice
  Journal Of Neural Transmission-Parkinsons Disease and Dementia Section, 1989
• Lack of tolerance to nicotine-induced dopamine release in the nucleus accumbens
  European Journal of Pharmacology, 1989
• Drugs abused by humans preferentially increase synaptic dopamine concentrations in the mesolimbic system of freely moving rats.
  Proceedings of the National Academy of Sciences, 1988