Macrophage Colony-stimulating Factor Augments β-Amyloid-induced Interleukin-1, Interleukin-6, and Nitric Oxide Production by Microglial Cells
Open Access
- 1 August 1998
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 273 (33) , 20967-20971
- https://doi.org/10.1074/jbc.273.33.20967
Abstract
No abstract availableKeywords
This publication has 58 references indexed in Scilit:
- CSF-I signal transaction: what is of functional significance?Immunology Today, 1997
- Microglial Cells Internalize Aggregates of the Alzheimer's Disease Amyloid β-Protein Via a Scavenger ReceptorNeuron, 1996
- RAGE and amyloid-β peptide neurotoxicity in Alzheimer's diseaseNature, 1996
- Cytokines in inflammatory brain lesions: helpful and harmfulTrends in Neurosciences, 1996
- Amyloid β Protein Primes Cultured Rat Microglial Cells for an Enhanced Phorbol 12‐Myristate 13‐Acetate‐Induced Respiratory Burst ActivityJournal of Neurochemistry, 1996
- Selective Killing of Cholinergic Neurons by Microglial Activation in Basal Forebrain Mixed Neuronal/Glial CulturesBiochemical and Biophysical Research Communications, 1995
- Nitric oxide up‐regulates the release of inflammatory mediators by mouse macrophagesEuropean Journal of Immunology, 1995
- Activation of Macrophages by Alzheimer β Amyloid PeptideBiochemical and Biophysical Research Communications, 1994
- Distinguishing between mouse IL-3 and IL-3 receptor-like (IL-5/GM-CSF receptor converter) mRNAs using the polymerase chain reaction methodJournal of Immunological Methods, 1992
- Interleukin‐6 and α‐2‐macroglobulin indicate an acute‐phase state in Alzheimer's disease corticesFEBS Letters, 1991