Failure of Tubular Reabsorptive Loads of Ascorbic Acid or Amino Acids to Affect Renal Handling of Sodium and Potassium

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Abstract
The renal tubular transport mechanisms for Na and K are affected neither by saturating the secretory mechanisms for carinamide, PAH or Diodrast, nor by loading the reabsorptive mechanisms for ascorbic acid or several amino acids. Since these procedures fail to alter either excretory rate for Na and K or percentage excretion of filtered Na and K, it would appear the tubular transport mechanisms for Na and K are distinct from those for Diodrast, PAH, carinamide, amino acids and ascorbic acid.