Vaccinia Virus E3L Protein Is an Inhibitor of the Interferon (IFN)-Induced 2-5A Synthetase Enzyme

This publication has 31 references indexed in Scilit:

• Selective extraction of polyoma DNA from infected mouse cell cultures
  Published by Elsevier ,2004
• A Study of the Interferon Antiviral Mechanism: Apoptosis Activation by the 2–5A System
  The Journal of Experimental Medicine, 1997
• Activation of the IFN-Inducible Enzyme RNase L Causes Apoptosis of Animal Cells
  Virology, 1997
• Inducible Expression of the 2-5A Synthetase/RNase L System Results in Inhibition of Vaccinia Virus Replication
  Virology, 1997
• When Two Strands Are Better Than One: The Mediators and Modulators of the Cellular Responses to Double-Stranded RNA
  Virology, 1996
• Identification of a novel serine/threonine kinase and a novel 15-kD protein as potential mediators of the gamma interferon-induced cell death.
  Genes & Development, 1995
• Interferon‐γ arrests proliferation and causes apoptosis in stromal cell/interleukin‐7‐dependent normal murine pre‐B cell lines and clones in vitro, but does not induce differentiation to surface immunoglobulin‐positive B cells
  European Journal of Immunology, 1993
• Identification of programmed cell death in situ via specific labeling of nuclear DNA fragmentation.
  The Journal of cell biology, 1992
• The E3L gene of vaccinia virus encodes an inhibitor of the interferon-induced, double-stranded RNA-dependent protein kinase.
  Proceedings of the National Academy of Sciences, 1992
• Eukaryotic transient-expression system based on recombinant vaccinia virus that synthesizes bacteriophage T7 RNA polymerase.
  Proceedings of the National Academy of Sciences, 1986