2-Methoxyestradiol, an Endogenous Metabolite of Estrogen, Enhances Apoptosis and β-Galactosidase Expression in Vascular Endothelial Cells
- 1 July 1998
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 248 (1) , 9-12
- https://doi.org/10.1006/bbrc.1998.8902
Abstract
No abstract availableKeywords
This publication has 21 references indexed in Scilit:
- The mammalian metabolite, 2-methoxyestradiol, affects p53 levels and apoptosis induction in transformed cells but not in normal cellsThe Journal of Steroid Biochemistry and Molecular Biology, 1997
- Estrogen-Receptor–Mediated Inhibition of Human Endothelial Cell ApoptosisCirculation, 1997
- A biomarker that identifies senescent human cells in culture and in aging skin in vivo.Proceedings of the National Academy of Sciences, 1995
- Accelerated growth and senescence of arterial endothelial cells expressing the small molecular weight heat‐shock protein HSP27The FASEB Journal, 1995
- Evidence that nitric oxide is an endogenous antiangiogenic mediatorBritish Journal of Pharmacology, 1994
- The endogenous oestrogen metabolite 2-methoxyoestradiol inhibits angiogenesis and suppresses tumour growthNature, 1994
- Apoptosis and the regulation of cell numbers in normal and neoplastic tissues: an overviewCancer and Metastasis Reviews, 1992
- Phenol red in tissue culture media is a weak estrogen: implications concerning the study of estrogen-responsive cells in culture.Proceedings of the National Academy of Sciences, 1986
- Identification and isolation of endothelial cells based on their increased uptake of acetylated-low density lipoprotein.The Journal of cell biology, 1984
- Analysis of nitrate, nitrite, and [15N]nitrate in biological fluidsAnalytical Biochemistry, 1982