Point Mutation in trkB Causes Loss of NT4-Dependent Neurons without Major Effects on Diverse BDNF Responses
Open Access
- 1 August 1998
- Vol. 21 (2) , 335-345
- https://doi.org/10.1016/s0896-6273(00)80543-7
Abstract
No abstract availableKeywords
This publication has 61 references indexed in Scilit:
- Abnormal Cerebellar Development and Foliation in BDNF−/− Mice Reveals a Role for Neurotrophins in CNS PatterningNeuron, 1997
- Signal transduction by the neutrophin receptorsCurrent Opinion in Cell Biology, 1997
- Neurotrophin switching: where does it stand?Current Opinion in Neurobiology, 1997
- Dissociation of NGF Induced Signal Transduction from Neurite Elongation by Expression of a Mutant Adaptor Protein v-Crk in PC12 CellsMolecular and Cellular Neuroscience, 1996
- Phosphotyrosine Residues in the Nerve‐Growth‐Factor Receptor (Trk‐A)European Journal of Biochemistry, 1995
- NT-4-mediated rescue of lateral geniculate neurons from effects of monocular deprivationNature, 1995
- Early events in neurotrophin signalling via Trk and p75 receptorsCurrent Opinion in Neurobiology, 1995
- A conserved amino-terminal Shc domain binds to phosphotyrosine motifs in activated receptors and phosphopeptidesCurrent Biology, 1995
- An Alternative to SH2 Domains for Binding Tyrosine-Phosphorylated ProteinsScience, 1994
- Targeted disruption of the trkB neurotrophin receptor gene results in nervous system lesions and neonatal deathCell, 1993