Eotaxin-2 and eotaxin-3 expression is associated with persistent eosinophilic bronchial inflammation in patients with asthma after allergen challenge

This publication has 45 references indexed in Scilit:

• Interleukin (IL)-4 and to a lesser extent either IL-13 or interferon-gamma regulate the production of eotaxin-2/CCL24 in nasal polyps
  Allergy, 2003
• REGULATION OF HUMAN EOTAXIN-3/CCL26 EXPRESSION: MODULATION BY CYTOKINES AND GLUCOCORTICOIDS
  Cytokine, 2002
• Allergen-induced impairment of bronchoprotective nitric oxide synthesis in asthma
  Journal of Allergy and Clinical Immunology, 2001
• Chemokines induce eosinophil degranulation through CCR-3
  Journal of Allergy and Clinical Immunology, 2000
• Repeatability of measures of inflammatory cell number in bronchial biopsies in atopic asthma
  European Respiratory Journal, 1997
• Eotaxin-2, a Novel CC Chemokine that Is Selective for the Chemokine Receptor CCR3, and Acts Like Eotaxin on Human Eosinophil and Basophil Leukocytes
  The Journal of Experimental Medicine, 1997
• Targeted Disruption of the Chemokine Eotaxin Partially Reduces Antigen-induced Tissue Eosinophilia
  The Journal of Experimental Medicine, 1997
• Cooperation between interleukin-5 and the chemokine eotaxin to induce eosinophil accumulation in vivo.
  The Journal of Experimental Medicine, 1995
• The Chemokine, Eotaxin, Activates Guinea-Pig Eosinophils in Vitro and Causes Their Accumulation into the Lung in Vivo
  Biochemical and Biophysical Research Communications, 1993
• Eosinophils, T-lymphocytes, mast cells, neutrophils, and macrophages in bronchial biopsy specimens from atopic subjects with asthma: Comparison with biopsy specimens from atopic subjects without asthma and normal control subjects and relationship to bronchial hyperresponsiveness
  Journal of Allergy and Clinical Immunology, 1991