Acquired B Antigen Disappearance byin vitvoAcetylation Associated with A1Activity Restoration

Abstract
The chemical acetylation of [human] RBC [red blood cells] bearing the acquired B antigen led to the disappearance of the agglutinability by anti-B and restored the A1 specificity. The same results are obtained using RBC transformed in vitro by a Clostridium Tertium filtrate, where a deacetylase was reported.