Mechanism of resistance of non-cycling mammalian cells to 4′-[9-acridinylamino]methanesulphon-m-anisidide: Role of DNA topoisomerase II in log- and plateau-phase CHO cells
- 1 March 1988
- journal article
- research article
- Published by Elsevier in Biochimica et Biophysica Acta (BBA) - Gene Structure and Expression
- Vol. 949 (3) , 264-272
- https://doi.org/10.1016/0167-4781(88)90151-0
Abstract
No abstract availableThis publication has 30 references indexed in Scilit:
- Proliferation dependence of topoisomerase II-mediated drug actionBiochemistry, 1986
- The production of topoisomerase II-mediated DNA cleavage in human leukemia cells predicts their susceptibility to 4′-(9-acridinylamino)methanesulfon-m-anisidide (m-AMSA)Biochemical and Biophysical Research Communications, 1986
- DNA topoisomerase I and II activities during cell proliferation and the cell cycle in cultured mouse embryo fibroblast (C3H 10T1/2) cellsExperimental Cell Research, 1985
- DNA topoisomerase activities in concanavalin A‐stimulated lymphocytesFEBS Letters, 1984
- Adriamycin-Induced DNA Damage Mediated by Mammalian DNA Topoisomerase IIScience, 1984
- Anti-cancer drugs which intercalate into DNA: How do they act?Trends in Biochemical Sciences, 1983
- DNA topoisomerases from rat liver: physiological variationsNucleic Acids Research, 1983
- Evaluation of AMSA in previously treated patients with acute leukemia: results of therapy in 109 adultsBlood, 1982
- Novel topologically knotted DNA from bacteriophage P4 capsids: studies with DNA topoisomerasesNucleic Acids Research, 1981
- A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye bindingAnalytical Biochemistry, 1976