Tumor Necrosis Factor α Inhibits Insulin-induced Mitogenic Signaling in Vascular Smooth Muscle Cells

This publication has 29 references indexed in Scilit:

• Proliferative Effect of Insulin on Cultured Smooth Muscle Cells From Rat Mesenteric Resistance Vessels
  American Journal of Hypertension, 1998
• Troglitazone enhances glucose uptake and inhibits mitogen-activated protein kinase in human aortic smooth muscle cells
  Atherosclerosis, 1998
• Inhibition of MAP kinase blocks insulin‐mediated DNA synthesis and transcriptional activation of c‐fos by Elk‐1 in vascular smooth muscle cells
  FEBS Letters, 1997
• Two Naturally Occurring Insulin Receptor Tyrosine Kinase Domain Mutants Provide Evidence That Phosphoinositide 3-Kinase Activation Alone Is Not Sufficient for the Mediation of Insulin's Metabolic and Mitogenic Effects
  Journal of Biological Chemistry, 1997
• Multiple Signaling Pathways Involved in the Metabolic Effects of Insulin
  The American Journal of Cardiology, 1997
• The IRS‐signalling system during insulin and cytokine action
  BioEssays, 1997
• Comparison of the insulin and insulin-like growth factor 1 mitogenic intracellular signaling pathways.
  Endocrinology, 1996
• IRS-1-Mediated Inhibition of Insulin Receptor Tyrosine Kinase Activity in TNF-α- and Obesity-Induced Insulin Resistance
  Science, 1996
• Differential inhibitory effects of antidiabetic drugs on arterial smooth muscle cell proliferation
  American Journal of Hypertension, 1996
• Tumor necrosis factor alpha inhibits signaling from the insulin receptor.
  Proceedings of the National Academy of Sciences, 1994