Opposite effects of TGF-β1 on rapidly- and slowly-triggered excitotoxic injury
- 1 March 1996
- journal article
- research article
- Published by Elsevier in Neuropharmacology
- Vol. 35 (3) , 249-256
- https://doi.org/10.1016/0028-3908(96)00001-9
Abstract
No abstract availableKeywords
This publication has 38 references indexed in Scilit:
- Transcriptional or translational inhibition blocks low dose NMDA-mediated cell deathNeuroReport, 1995
- Cleavage of DNA to Large Kilobase Pair Fragments Occurs in Some Forms of Necrosis as Well as ApoptosisBiochemical and Biophysical Research Communications, 1995
- Death from inside out: an overviewPhilosophical Transactions Of The Royal Society B-Biological Sciences, 1994
- Basic fibroblast growth factor protects cerebellar neurons in primary culture from NMDA and non‐NMDA receptor mediated neurotoxicityFEBS Letters, 1993
- Brain‐derived Neurotrophic Factor is a Survival Factor for Cultured Rat Cerebellar Granule Neurons and Protects them Against Glutamate‐induced NeurotoxicityEuropean Journal of Neuroscience, 1993
- GDNF: a Glial Cell Line-Derived Neurotrophic Factor for Midbrain Dopaminergic NeuronsScience, 1993
- Effects of transforming growth factor-beta on murine astrocyte glutamine synthetase activity. Implications in neuronal injury.Journal of Clinical Investigation, 1992
- Identification of programmed cell death in situ via specific labeling of nuclear DNA fragmentation.The Journal of cell biology, 1992
- Glutamate triggers internucleosomal DNA cleavage in neuronal cellsBiochemical and Biophysical Research Communications, 1991
- Neurotoxicity of excitatory amino acid receptor agonists in rat cerebellar slices: Dependence on calcium concentrationNeuroscience Letters, 1986