Cupric nitrilotriacetate induces oxidative DNA damage and apoptosis in human leukemia HL-60 cells
- 1 September 1998
- journal article
- Published by Elsevier in Free Radical Biology & Medicine
- Vol. 25 (4-5) , 568-575
- https://doi.org/10.1016/s0891-5849(98)00088-4
Abstract
No abstract availableKeywords
This publication has 44 references indexed in Scilit:
- Oxidative stress as a mediator of apoptosisPublished by Elsevier ,2002
- A role for metals and free radicals in the induction of apoptosis in thymocytesFEBS Letters, 1994
- bcl-x, a bcl-2-related gene that functions as a dominant regulator of apoptotic cell deathCell, 1993
- N‐Acetylcysteine inhibits apoptosis and decreases viral particles in HIV‐chronically infected U937 cellsFEBS Letters, 1993
- Programmed cell death: Apoptosis and oncogenesisCell, 1991
- DNA damage by oxygen‐derived species Its mechanism and measurement in mammalian systemsFEBS Letters, 1991
- Tumor necrosis factor induced DNA fragmentation of HL-60 cellsBiochemical and Biophysical Research Communications, 1988
- Site-specific cleavage of single-stranded DNAs at unique sites by a copper-dependent redox reactionNature, 1988
- Oral administration of the renal carcinogen, potassium bromate, specifically produces 8-hydroxydeoxyguanosine in rat target organ DNACarcinogenesis: Integrative Cancer Research, 1987
- Formation of 8-hydroxyguanine moiety in cellular DNA by agents producing oxygen radicals and evidence for its repairCarcinogenesis: Integrative Cancer Research, 1986