Abstract
Onset of schedule-induced polydipsia (SIP) is related to adrenal gland weight. Adrenalectomy, but not demedullation, hastened the emergence of SIP, and exogenous corticosterone administration reversed this effect. Hippocampal lesions were followed by a rapid and uniform release of SIP. None of the above manipulations influenced normal (home-cage) drinking. The hippocampus and the adrenal cortex apparently interact and the equilibrium established within this system is reflected, for any particular rat, in its adjunctive behavior.

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