Celiac Disease: Caught Between a Rock and a Hard Place

This publication has 57 references indexed in Scilit:

• The Molecular Basis for Oat Intolerance in Patients with Celiac Disease
  PLoS Medicine, 2004
• Genomewide search and association studies in a Finnish celiac disease population: Identification of a novel locus and replication of the HLA and CTLA4 loci
  American Journal of Medical Genetics Part A, 2004
• Coordinated Induction by IL15 of a TCR-Independent NKG2D Signaling Pathway Converts CTL into Lymphokine-Activated Killer Cells in Celiac Disease
  Immunity, 2004
• A Direct Role for NKG2D/MICA Interaction in Villous Atrophy during Celiac Disease
  Immunity, 2004
• Gliadin stimulates human monocytes to production of IL‐8 and TNF‐α through a mechanism involving NF‐κB
  FEBS Letters, 2004
• Characterization of cereal toxicity for celiac disease patients based on protein homology in grains 1 1The authors thank Drs. R. R. P. de Vries and R. Offringa for critical reading of the manuscript, A. de Ru for mass spectrometric analysis, and W. Benckhuijsen for peptide synthesis.
  Gastroenterology, 2003
• Role of interferon alpha in promoting T helper cell type 1 responses in the small intestine in coeliac disease
  Gut, 2001
• Dermatitis herpetiformis and celiac disease are both primarily associated with the HLA‐DQ (α1*0501, (β1*02) or the HLA‐DQ (α1*03, (β1*0302) heterodimers
  Tissue Antigens, 1997
• T cells from the small intestinal Mucosa of a DR4, DQ7/DR4. DQ8 celiac disease patient preferentially recognize gliadin when presented by DQ8
  Human Immunology, 1994
• Gliadin-specific, HLA-DQ(alpha 1*0501,beta 1*0201) restricted T cells isolated from the small intestinal mucosa of celiac disease patients.
  The Journal of Experimental Medicine, 1993