Inflammatory responses to ischemia, and reperfusion in skeletal muscle
- 1 January 1998
- journal article
- review article
- Published by Springer Nature in Molecular and Cellular Biochemistry
- Vol. 179 (1/2) , 169-187
- https://doi.org/10.1023/a:1006832207864
Abstract
Skeletal muscle ischemia and reperfusion is now recognized as one form of acute inflammation in which activated leukocytes play a key role. Although restoration of flow is essential in alleviating...Keywords
This publication has 94 references indexed in Scilit:
- Inhibition of polymorphonuclear leukocyte adherence suppresses no-reflow after focal cerebral ischemia in baboons.Stroke, 1992
- Polymorphonuclear leukocytes occlude capillaries following middle cerebral artery occlusion and reperfusion in baboons.Stroke, 1991
- Reduction of central nervous system ischemic injury in rabbits using leukocyte adhesion antibody treatment.Stroke, 1991
- The effect of ischemia/reperfusion on adenine nucleotide metabolism and xanthine oxidase production in skeletal muscleJournal of Vascular Surgery, 1990
- Desferrioxamine as a lipid chain‐breaking antioxidant in sickle erythrocyte membranesFEBS Letters, 1990
- Tissue Destruction by NeutrophilsNew England Journal of Medicine, 1989
- Xanthine oxidase produces hydrogen peroxide which contributes to reperfusion injury of ischemic, isolated, perfused rat hearts.Journal of Clinical Investigation, 1988
- Allopurinol and oxypurinol are hydroxyl radical scavengersFEBS Letters, 1987
- Oxygen-Derived Free Radicals in Postischemic Tissue InjuryNew England Journal of Medicine, 1985
- Reassessment of cerebral capillary changes in acute global ischemia and their relationship to the "no-reflow phenomenon".Stroke, 1977