Infliximab induces apoptosis in monocytes from patients with chronic active Crohn's disease by using a caspase-dependent pathway
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- 1 November 2001
- journal article
- Published by Elsevier in Gastroenterology
- Vol. 121 (5) , 1145-1157
- https://doi.org/10.1053/gast.2001.28702
Abstract
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This publication has 48 references indexed in Scilit:
- Infliximab for the Treatment of Fistulas in Patients with Crohn's DiseaseNew England Journal of Medicine, 1999
- Tumor necrosis factor α antibody (infliximab) therapy profoundly down-regulates the inflammation in Crohn's ileocolitisPublished by Elsevier ,1999
- Caspase-3 Is Required for DNA Fragmentation and Morphological Changes Associated with ApoptosisJournal of Biological Chemistry, 1998
- FLICE, A Novel FADD-Homologous ICE/CED-3–like Protease, Is Recruited to the CD95 (Fas/APO-1) Death-Inducing Signaling ComplexCell, 1996
- Involvement of MACH, a Novel MORT1/FADD-Interacting Protease, in Fas/APO-1- and TNF Receptor–Induced Cell DeathCell, 1996
- Construction and initial characterization of a mouse-human chimeric anti-TNF antibodyMolecular Immunology, 1993
- ApoptosisImmunology Today, 1993
- Tumor necrosis factor-?, interleukin-1?, and interleukin-6 expression in inflammatory bowel diseaseDigestive Diseases and Sciences, 1992
- The Pathophysiology of Tumor Necrosis FactorsAnnual Review of Immunology, 1992
- Lymphocytes play the music but the macrophage calls the tuneImmunology Today, 1991