p53 Mediates the Accelerated Onset of Senescence of Endothelial Progenitor Cells in Diabetes
Open Access
- 1 February 2006
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 281 (7) , 4339-4347
- https://doi.org/10.1074/jbc.m509293200
Abstract
No abstract availableKeywords
This publication has 53 references indexed in Scilit:
- Absence of p21 Waf1/Cip1/Sdi1 Modulates Macrophage Differentiation and Inflammatory Response and Protects Against AtherosclerosisCirculation, 2004
- RAGE‐ and TGF‐ β receptor‐mediated signals converge on STAT5 and p21wafto control cell‐cycle progression of mesangial cells: a possible role in the development and progression of diabetic nephropathyThe FASEB Journal, 2004
- Genes that act downstream of DAF-16 to influence the lifespan of Caenorhabditis elegansNature, 2003
- Mildly oxidized LDL particle subspecies are distinct in their capacity to induce apoptosis in endothelial cells: role of lipid hydroperoxidesThe FASEB Journal, 2002
- Diabetic LDL inhibits cell-cycle progression via STAT5B and p21wafJournal of Clinical Investigation, 2002
- Regulation ofp53downstream genesSeminars in Cancer Biology, 1998
- Adenovirus-mediated over-expression of the cyclin/cyclin-dependent kinase inhibitor, p21 inhibits vascular smooth muscle cell proliferation and neointima formation in the rat carotid artery model of balloon angioplasty.Journal of Clinical Investigation, 1995
- Lipoprotein metabolism in diabetesCurrent Opinion in Lipidology, 1994
- The pathogenesis of atherosclerosis: a perspective for the 1990sNature, 1993
- Role of Oxidative Stress in Development of Complications in DiabetesDiabetes, 1991