c-Myc Can Induce DNA Damage, Increase Reactive Oxygen Species, and Mitigate p53 Function
Top Cited Papers
- 1 May 2002
- journal article
- Published by Elsevier in Molecular Cell
- Vol. 9 (5) , 1031-1044
- https://doi.org/10.1016/s1097-2765(02)00520-8
Abstract
No abstract availableKeywords
This publication has 74 references indexed in Scilit:
- The evolution of diverse biological responses to DNA damage: insights from yeast and p53Nature Cell Biology, 2001
- Gene-target recognition among members of the Myc superfamily and implications for oncogenesisNature Genetics, 2000
- ATM: A mediator of multiple responses to genotoxic stressOncogene, 1999
- Disruption of Redox Homeostasis in the Transforming Growth Factor-α/c-myc Transgenic Mouse Model of Accelerated HepatocarcinogenesisJournal of Biological Chemistry, 1998
- Oncogenic ras Provokes Premature Cell Senescence Associated with Accumulation of p53 and p16INK4aCell, 1997
- Production of hydrogen peroxide by transforming growth factor-beta 1 and its involvement in induction of egr-1 in mouse osteoblastic cells.The Journal of cell biology, 1994
- Effect of chromosome size on aberration levels caused by gamma radiation as detected by fluorescence in situ hybridizationCytogenetic and Genome Research, 1994
- Altered cell cycle arrest and gene amplification potential accompany loss of wild-type p53Published by Elsevier ,1992
- Wild-type p53 restores cell cycle control and inhibits gene amplification in cells with mutant p53 allelesCell, 1992
- Induction of apoptosis in fibroblasts by c-myc proteinCell, 1992