Oncogenic Ras Enhances NF-κB Transcriptional Activity through Raf-dependent and Raf-independent Mitogen-activated Protein Kinase Signaling Pathways

This publication has 46 references indexed in Scilit:

• NF-κB AND REL PROTEINS: Evolutionarily Conserved Mediators of Immune Responses
  Annual Review of Immunology, 1998
• p38 and Extracellular Signal-regulated Kinase Mitogen-activated Protein Kinase Pathways Are Required for Nuclear Factor-κB p65 Transactivation Mediated by Tumor Necrosis Factor
  Journal of Biological Chemistry, 1998
• Identification and Characterization of an IκB Kinase
  Cell, 1997
• The Transcriptional Activity of NF-κB Is Regulated by the IκB-Associated PKAc Subunit through a Cyclic AMP–Independent Mechanism
  Cell, 1997
• Emerging from the Pak: the p21-activated protein kinase family
  Trends in Cell Biology, 1997
• Involvement of Egr-1/RelA Synergy in Distinguishing T Cell Activation from Tumor Necrosis Factor-α–induced NF-κB1 Transcription
  The Journal of Experimental Medicine, 1997
• THE NF-κB AND IκB PROTEINS: New Discoveries and Insights
  Annual Review of Immunology, 1996
• Rapid induction of heparin-binding epidermal growth factor/diphtheria toxin receptor expression by Raf and Ras oncogenes.
  Genes & Development, 1995
• Proteins regulating Ras and its relatives
  Nature, 1993
• Serum-, TPA-, and Ras-induced expression from Ap-1/Ets-driven promoters requires Raf-1 kinase.
  Genes & Development, 1992