CTG/CAG Repeat Instability Is Modulated by the Levels of Human DNA Ligase I and Its Interaction with Proliferating Cell Nuclear Antigen

This publication has 100 references indexed in Scilit:

• Long Patch Base Excision Repair Proceeds via Coordinated Stimulation of the Multienzyme DNA Repair Complex
  Journal of Biological Chemistry, 2009
• Mechanism of replication machinery assembly as revealed by the DNA ligase–PCNA–DNA complex architecture
  Proceedings of the National Academy of Sciences, 2009
• Division of Labor at the Eukaryotic Replication Fork
  Published by Elsevier ,2008
• A case‐control study of the association of the polymorphisms and haplotypes of DNA ligase I with lung and upper‐aerodigestive‐tract cancers
  International Journal of Cancer, 2008
• Sequential recruitment of the repair factors during NER: the role of XPG in initiating the resynthesis step
  The EMBO Journal, 2007
• OGG1 initiates age-dependent CAG trinucleotide expansion in somatic cells
  Nature, 2007
• The C-terminal domain of yeast PCNA is required for physical and functional interactions with Cdc9 DNA ligase
  Nucleic Acids Research, 2007
• Haploinsufficiency of yeast FEN1 causes instability of expanded CAG/CTG tracts in a length-dependent manner
  Gene, 2007
• Proliferating cell nuclear antigen loaded onto double-stranded DNA: dynamics, minor groove interactions and functional implications
  Nucleic Acids Research, 2006
• Differential recruitment of DNA Ligase I and III to DNA repair sites
  Nucleic Acids Research, 2006