Plasma proteins containing damaged L-isoaspartyl residues are increased in uremia: Implications for mechanism

This publication has 37 references indexed in Scilit:

• Increased methyl esterification of altered aspartyl residues in erythrocyte membrane proteins in response to oxidative stress
  European Journal of Biochemistry, 2000
• Spontaneous Deamidation and Isomerization of Asn108 in Prion Peptide 106–126 and in Full-Length Prion Protein
  Biochemical and Biophysical Research Communications, 1999
• Measurement of Altered Aspartyl Residues in the Scrapie Associated Form of Prion Protein
  Biochemical and Biophysical Research Communications, 1998
• Cytoskeletal behaviour in spectrin and in band 3 deficient spherocytic red cells: evidence for a differentiated splenic conditioning role
  British Journal of Haematology, 1996
• Protein damage and methylation-mediated repair in the erythrocyte
  Biochemical Journal, 1995
• Increased Membrane-Protein Methylation in Hereditary Spherocytosis. A Marker of Cytoskeletal Disarray
  European Journal of Biochemistry, 1995
• beta-Amyloid-(1-42) is a major component of cerebrovascular amyloid deposits: implications for the pathology of Alzheimer disease.
  Proceedings of the National Academy of Sciences, 1993
• Structural alterations in the peptide backbone of beta-amyloid core protein may account for its deposition and stability in Alzheimer's disease.
  Journal of Biological Chemistry, 1993
• Repair of isopeptide bonds by protein carboxyl O-methyltransferase: seminal ribonuclease as a model system
  Biochemistry, 1988
• Increased methyl esterification of membrane proteins in aged red‐blood cells
  European Journal of Biochemistry, 1983