Evidence that lipopolysaccharide-induced cell death is mediated by accumulation of reactive oxygen species and activation of p38 in rat cortex and hippocampus
- 1 December 2003
- journal article
- research article
- Published by Elsevier in Experimental Neurology
- Vol. 184 (2) , 794-804
- https://doi.org/10.1016/s0014-4886(03)00301-7
Abstract
No abstract availableKeywords
This publication has 56 references indexed in Scilit:
- •NO and Oxyradical Metabolism in New Cell Lines of Rat Brain Capillary Endothelial Cells Forming the Blood–Brain BarrierMicrovascular Research, 2001
- Oxidative stress induced-neurodegenerative diseases: the need for antioxidants that penetrate the blood brain barrierPublished by Elsevier ,2001
- Modulation of Protein Kinase Activity and Gene Expression by Reactive Oxygen Species and Their Role in Vascular Physiology and PathophysiologyArteriosclerosis, Thrombosis, and Vascular Biology, 2000
- Cell Proliferation and Apoptosis in the Immune System in the ElderlyImmunologic Research, 2000
- The neuronal death induced by endotoxic shock but not that induced by excitatory amino acids requires TNF‐αEuropean Journal of Neuroscience, 1998
- Oxidative stress and apoptosis in neurodegenerationJournal of the Neurological Sciences, 1996
- Involvement of reactive oxygen intermediates in cyclooxygenase-2 expression induced by interleukin-1, tumor necrosis factor-alpha, and lipopolysaccharide.Journal of Clinical Investigation, 1995
- Receptors for interleukin-1 (α and β) in mouse brain: Mapping and neuronal localization in hippocampusNeuroscience, 1991
- Protection of Neocortical Tissue Prisms from Freeze‐Thaw Injury by Dimethyl SulphoxideJournal of Neurochemistry, 1981
- A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye bindingAnalytical Biochemistry, 1976