Impaired Neuronal Plasticity in Transgenic Mice Expressing Human Apolipoprotein E4 Compared to E3 in a Model of Entorhinal Cortex Lesion

This publication has 58 references indexed in Scilit:

• Expression of Human Apolipoprotein E4 in Neurons Causes Hyperphosphorylation of Protein Tau in the Brains of Transgenic Mice
  The American Journal of Pathology, 2000
• Intraventricular Infusion of Apolipoprotein E Ameliorates Acute Neuronal Damage after Global Cerebral Ischemia in Mice
  Journal of Cerebral Blood Flow & Metabolism, 2000
• A Quantitative and Immunohistochemical Study on Apolipoprotein E in Brain Tissue in Alzheimer’s Disease
  Dementia and Geriatric Cognitive Disorders, 1999
• Specific Regional Transcription of Apolipoprotein E in Human Brain Neurons
  The American Journal of Pathology, 1999
• The neurobiology of apolipoproteins and their receptors in the CNS and Alzheimer's disease
  Brain Research Reviews, 1998
• Evidence for Normal Aging of the Septo-Hippocampal Cholinergic System in apoE (−/−) Mice but Impaired Clearance of Axonal Degeneration Products Following Injury
  Experimental Neurology, 1998
• Epidemiological, Clinical, and Neuropathological Study of Apolipoprotein E Genotype in Alzheimer's Disease
  Annals of the New York Academy of Sciences, 1996
• Apolipoprotein E-containing High Density Lipoprotein Promotes Neurite Outgrowth and Is a Ligand for the Low Density Lipoprotein Receptor-related Protein
  Published by Elsevier ,1996
• Deposition of apolipoproteins E and J in senile plaques is topographically determined in both Alzheimer's disease and Down's syndrome brain
  Brain Research, 1995
• Synaptic reorganization within the human central nervous system following injury
  Journal of Neurosurgery, 1989