MODULATION OF GLUTAMATE RECEPTORS BY CALCIUM/CALMODULIN-DEPENDENT PROTEIN KINASE II
- 1 April 1996
- journal article
- review article
- Published by Elsevier in Neurochemistry International
- Vol. 28 (4) , 359-361
- https://doi.org/10.1016/0197-0186(95)00098-4
Abstract
No abstract availableThis publication has 24 references indexed in Scilit:
- Long-term potentiation is associated with an increased activity of Ca2+/calmodulin-dependent protein kinase II.Published by Elsevier ,2021
- CaM KINASE II IN LONG-TERM POTENTIATIONNeurochemistry International, 1996
- A synaptic model of memory: long-term potentiation in the hippocampusNature, 1993
- Ca2+ Entry via postsynaptic voltage-sensitive Ca2+ channels can transiently potentiate excitatory synaptic transmission in the hippocampusNeuron, 1992
- NEURONAL Ca2+/CALMODULIN-DEPENDENT PROTEIN KINASESAnnual Review of Biochemistry, 1992
- Perforated synapses and plasticityMolecular Neurobiology, 1991
- Effects of KN-62, a specific inhibitor of calcium/calmodulin-dependent protein kinase II, on long-term potentiation in the rat hippocampusNeuroscience Letters, 1991
- Temporally distinct pre- and post-synaptic mechanisms maintain long-term potentiationNature, 1989
- Evidence that the major postsynaptic density protein is a component of a Ca2+/calmodulin-dependent protein kinase.Proceedings of the National Academy of Sciences, 1984
- Biochemical and immunochemical evidence that the "major postsynaptic density protein" is a subunit of a calmodulin-dependent protein kinase.Proceedings of the National Academy of Sciences, 1983