Conditional deletion of the TGF-β type II receptor in Col2a expressing cells results in defects in the axial skeleton without alterations in chondrocyte differentiation or embryonic development of long bones
- 11 September 2004
- journal article
- research article
- Published by Elsevier in Developmental Biology
- Vol. 276 (1) , 124-142
- https://doi.org/10.1016/j.ydbio.2004.08.027
Abstract
No abstract availableKeywords
This publication has 68 references indexed in Scilit:
- Mechanisms of TGF-β Signaling from Cell Membrane to the NucleusCell, 2003
- A kinase-inactive type II TGFβ receptor impairs BMP signaling in human breast cancer cellsBiochemical and Biophysical Research Communications, 2003
- Consequences of knocking out BMP signaling in the mouseGenesis, 2002
- Conditional inactivation of the TGF‐β type II receptor using Cre:LoxGenesis, 2002
- Bone abnormalities in latent TGF-β binding protein (Ltbp)-3–null mice indicate a role for Ltbp-3 in modulating TGF-β bioavailabilityThe Journal of cell biology, 2002
- Intracellular signalling: The Mad way to do itCurrent Biology, 1996
- BMP-7 is an inducer of nephrogenesis, and is also required for eye development and skeletal patterning.Genes & Development, 1995
- Mechanism of activation of the TGF-β receptorNature, 1994
- Targeted disruption of the mouse transforming growth factor-β1 gene results in multifocal inflammatory diseaseNature, 1992
- Differential staining of cartilage and bone in whole mouse fetuses by alcian blue and alizarin red STeratology, 1980