Candidate gene association studies of the α4 (CHRNA4) and β2 (CHRNB2) neuronal nicotinic acetylcholine receptor subunit genes in Alzheimer's disease
- 25 March 2004
- journal article
- research article
- Published by Elsevier in Neuroscience Letters
- Vol. 358 (2) , 142-146
- https://doi.org/10.1016/j.neulet.2004.01.016
Abstract
No abstract availableKeywords
This publication has 21 references indexed in Scilit:
- Expanding the association between the APOE gene and the risk of Alzheimer's disease: possible roles for APOE promoter polymorphisms and alterations in APOE transcriptionJournal of Neurochemistry, 2003
- Are interleukin-1 gene polymorphisms risk factors or disease modifiers in AD?Neurology, 2002
- Genes, models and Alzheimer's diseaseTrends in Genetics, 2001
- Decreased Protein Levels of Nicotinic Receptor Subunits in the Hippocampus and Temporal Cortex of Patients with Alzheimer’s DiseaseJournal of Neurochemistry, 2000
- α4 but Not α3 and α7 Nicotinic Acetylcholine Receptor Subunits Are Lost from the Temporal Cortex in Alzheimer's DiseaseJournal of Neurochemistry, 1999
- The cholinergic hypothesis of Alzheimer's disease: a review of progressJournal of Neurology, Neurosurgery & Psychiatry, 1999
- Stimulation of α4β2 nicotinic acetylcholine receptors inhibits β-amyloid toxicityBrain Research, 1998
- Nicotinic receptor stimulation protects neurons against β‐amyloid toxicityAnnals of Neurology, 1997
- The Cholinergic Hypothesis of Geriatric Memory DysfunctionScience, 1982
- “Mini-mental state”Journal of Psychiatric Research, 1975