Progressive neuronal loss and behavioral impairments of transgenic C57BL/6 inbred mice expressing the carboxy terminus of amyloid precursor protein
- 1 April 2006
- journal article
- Published by Elsevier in Neurobiology of Disease
- Vol. 22 (1) , 10-24
- https://doi.org/10.1016/j.nbd.2005.09.011
Abstract
No abstract availableKeywords
This publication has 67 references indexed in Scilit:
- Progressive cognitive impairment and anxiety induction in the absence of plaque deposition in C57BL/6 inbred mice expressing transgenic amyloid precursor proteinJournal of Neuroscience Research, 2004
- Inhibition of histone deacetylation enhances the neurotoxicity induced by the c‐terminal fragments of amyloid precursor proteinJournal of Neuroscience Research, 2003
- A CBP Binding Transcriptional Repressor Produced by the PS1/ϵ-Cleavage of N-Cadherin Is Inhibited by PS1 FAD MutationsCell, 2003
- Transgenic mice expressing the human C99 terminal fragment of βAPP: effects on spatial learning, exploration, anxiety, and motor coordinationExperimental Gerontology, 2002
- APP Processing and Synaptic Plasticity in Presenilin-1 Conditional Knockout MiceNeuron, 2001
- Presenilin 1 Regulates the Processing of β-Amyloid Precursor Protein C-Terminal Fragments and the Generation of Amyloid β-Protein in Endoplasmic Reticulum and GolgiBiochemistry, 1998
- Neurodegeneration and Gliosis in Transgenic Mice Overexpressing a Carboxy-Terminal Fragment of Alzheimer Amyloid-β Protein PrecursorDementia and Geriatric Cognitive Disorders, 1997
- Presenilin 1 is required for Notch 1 and Dll1 expression in the paraxial mesodermNature, 1997
- Transgenic mice expressing APP-C100 in the brainNeurobiology of Aging, 1996
- The Bcl-2 family of proteins, and the regulation of neuronal survivalTrends in Neurosciences, 1995