Nerve growth factor treatment or cAMP elevation reduces Ca2+/calmodulin-dependent protein kinase III activity in PC12 cells.
Open Access
- 1 October 1987
- journal article
- abstracts
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 262 (29) , 14265-14272
- https://doi.org/10.1016/s0021-9258(18)47933-1
Abstract
No abstract availableKeywords
This publication has 51 references indexed in Scilit:
- Calcium‐Dependent Nerve Growth Factor‐Stimulated Hydrolysis of Phosphoinositides in PC12 CellsJournal of Neurochemistry, 1987
- Nerve growth factor action is mediated by cyclic AMP- and Ca+2/phospholipid-dependent protein kinases.The Journal of cell biology, 1986
- Nerve growth factor does not activate Na+/H+ exchange in PC12 pheochromocytoma cellsJournal of Cellular Physiology, 1985
- Interleukin-2 stimulates association of protein kinase C with plasma membraneNature, 1985
- Ionic responses and growth stimulation induced by nerve growth factor and epidermal growth factor in rat pheochromocytoma (PC12) cells.The Journal of cell biology, 1983
- The role of protein phosphorylation in neural and hormonal control of cellular activityNature, 1982
- The Nerve Growth Factor: Biochemistry, Synthesis, and Mechanism of ActionAnnual Review of Neuroscience, 1980
- Troponin C-like proteins (calmodulins) from mammalian smooth muscle and other tissuesBiochemical Journal, 1979
- A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye bindingAnalytical Biochemistry, 1976
- Identification of a calcium-binding protein as a calcium-dependent regulator of brain adenylate cyclase.Proceedings of the National Academy of Sciences, 1975