MEC1-Dependent Redistribution of the Sir3 Silencing Protein from Telomeres to DNA Double-Strand Breaks
- 1 May 1999
- Vol. 97 (5) , 609-620
- https://doi.org/10.1016/s0092-8674(00)80772-2
Abstract
No abstract availableKeywords
This publication has 61 references indexed in Scilit:
- Yeast cell-type regulation of DNA repairNature, 1999
- The hMre11/hRad50 Protein Complex and Nijmegen Breakage Syndrome: Linkage of Double-Strand Break Repair to the Cellular DNA Damage ResponseCell, 1998
- Components of the Ku-dependent non-homologous end-joining pathway are involved in telomeric length maintenance and telomeric silencingThe EMBO Journal, 1998
- The RAD5 gene product is involved in the avoidance of non-homologous end-joining of DNA double strand breaks in the yeast Saccharomyces cerevisiaeNucleic Acids Research, 1997
- DNA double-strand-break sensitivity, DNA replication, and cell cycle arrest phenotypes of Ku-deficient Saccharomyces cerevisiaeProceedings of the National Academy of Sciences, 1997
- Identification of a Saccharomyces cerevisiae Ku80 homologue: roles in DNA double strand break rejoining and in telomeric maintenanceNucleic Acids Research, 1996
- Yeast Origin Recognition Complex Functions in Transcription Silencing and DNA ReplicationScience, 1993
- Modifiers of position effect are shared between telomeric and silent mating-type loci in S. cerevisiaeCell, 1991
- Fibroblasts from Ataxia Telangiectasia (AT) and AT Heterozygotes Show an Enhanced Level of Residual DNA Double-strand Breaks after Low Dose-rate γ-irradiation as Assayed by Pulsed Field Gel ElectrophoresisInternational Journal of Radiation Biology, 1991
- Regulated expression of endonuclease EcoRI in Saccharomyces cerevisiae: nuclear entry and biological consequences.Proceedings of the National Academy of Sciences, 1985