Intracellular Alkalinization Suppresses Lovastatin-induced Apoptosis in HL-60 Cells through the Inactivation of a pH-dependent Endonuclease
Open Access
- 1 March 1995
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 270 (11) , 6235-6242
- https://doi.org/10.1074/jbc.270.11.6235
Abstract
No abstract availableKeywords
This publication has 49 references indexed in Scilit:
- Inhibition of Isoprenoid Biosynthesis Induces Apoptosis in Human Promyelocytic HL-60 CellsBiochemical and Biophysical Research Communications, 1994
- Different patterns of apoptosis of HL‐60 cells induced by cycloheximide and camptothecinJournal of Cellular Physiology, 1993
- Role of isoprenylation in intracellular pH regulation of granulocytesFEBS Letters, 1993
- Endonuclease activation during apoptosis: The role of cytosolic Ca2+ and pHBiochemical and Biophysical Research Communications, 1992
- Regulation of intracellular actin polymerization by prenylated cellular proteinsThe Journal of cell biology, 1992
- Programmed cell death: Apoptosis and oncogenesisCell, 1991
- Regulation of the mevalonate pathwayNature, 1990
- Intracellular Ca2+ potentiates Na+/H+ exchange and cell differentiation induced by phorbol ester in U937 cellsEuropean Journal of Biochemistry, 1989
- pH homeostasis in promyelocytic leukemic HL60 cells.The Journal of general physiology, 1988
- Ethylisopropyl-amiloride: A new and highly potent derivative of amiloride for the inhibition of the Na+H+ exchange system in various cell typesBiochemical and Biophysical Research Communications, 1983