Transactivation of cyclin E gene by EWS-Fli1 and antitumor effects of cyclin dependent kinase inhibitor on Ewing's family tumor cells
Open Access
- 10 June 2005
- journal article
- research article
- Published by Wiley in International Journal of Cancer
- Vol. 116 (3) , 385-394
- https://doi.org/10.1002/ijc.21010
Abstract
No abstract availableKeywords
This publication has 37 references indexed in Scilit:
- Identification of p21 as a Direct Target of EWS-Fli1 Oncogenic Fusion ProteinPublished by Elsevier ,2003
- Alteration of Mesodermal Cell Differentiation by EWS/FLI-1, the Oncogene Implicated in Ewing's SarcomaMolecular and Cellular Biology, 2003
- High-dose chemotherapy and autologous peripheral blood stem-cell transfusion after conventional chemotherapy for patients with high-risk Ewing’s tumorsJournal of Orthopaedic Science, 2002
- Sp1 as G1 cell cycle phase specific transcription factor in epithelial cellsOncogene, 2002
- Loss of p16 pathways stabilizes EWS/FLI1 expression and complements EWS/FLI1 mediated transformationOncogene, 2001
- Biology of EWS/ETS fusions in Ewing's family tumorsOncogene, 2001
- Analysis of the expression of cell cycle regulators in Ewing cell lines: EWS-FLI-1 modulates p57KIP2 and c-Myc expressionOncogene, 2001
- Ets transcription factors cooperate with Sp1 to activate the human Tenascin-C promoterOncogene, 1999
- Principles of CDK regulationNature, 1995
- G1 phase progression: Cycling on cueCell, 1994