DNA polymerase η reduces the γ-H2AX response to psoralen interstrand crosslinks in human cells
- 17 November 2007
- journal article
- Published by Elsevier in Experimental Cell Research
- Vol. 314 (4) , 887-895
- https://doi.org/10.1016/j.yexcr.2007.10.031
Abstract
No abstract availableKeywords
Funding Information
- National Institutes of Health (CA105958)
This publication has 47 references indexed in Scilit:
- Cockayne syndrome exhibits dysregulation of p21 and other gene products that may be independent of transcription-coupled repairNeuroscience, 2007
- hMutSβ Is Required for the Recognition and Uncoupling of Psoralen Interstrand Cross-Links In VitroMolecular and Cellular Biology, 2002
- S. cerevisiae has three pathways for DNA interstrand crosslink repairMutation Research/DNA Repair, 2001
- Repair of DNA interstrand cross-linksMutation Research/DNA Repair, 2001
- Involvement of Nucleotide Excision Repair in a Recombination-Independent and Error-Prone Pathway of DNA Interstrand Cross-Link RepairMolecular and Cellular Biology, 2001
- Domain structure, localization, and function of DNA polymerase η, defective in xeroderma pigmentosum variant cellsGenes & Development, 2001
- P53 Binding Protein 1 (53bp1) Is an Early Participant in the Cellular Response to DNA Double-Strand BreaksThe Journal of cell biology, 2000
- Defining the Roles of Nucleotide Excision Repair and Recombination in the Repair of DNA Interstrand Cross-Links in Mammalian CellsMolecular and Cellular Biology, 2000
- DNA Replication Is Required To Elicit Cellular Responses to Psoralen-Induced DNA Interstrand Cross-LinksMolecular and Cellular Biology, 2000
- DNA Interstrand Cross-Links Induce Futile Repair Synthesis in Mammalian Cell ExtractsMolecular and Cellular Biology, 2000