Cell death by oxidative stress and ascorbic acid regeneration in human neuroectodermal cell lines

This publication has 20 references indexed in Scilit:

• Bcl-2 functions in an antioxidant pathway to prevent apoptosis
  Cell, 1993
• Bcl-2-deficient mice demonstrate fulminant lymphoid apoptosis, polycystic kidneys, and hypopigmented hair
  Cell, 1993
• Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programed cell death
  Cell, 1993
• bcl-x, a bcl-2-related gene that functions as a dominant regulator of apoptotic cell death
  Cell, 1993
• Multiple cell cycle access to the apoptotic death programme in human neuroblastoma cells
  FEBS Letters, 1993
• Social controls on cell survival and cell death
  Nature, 1992
• Bcl-2 gene promotes haemopoietic cell survival and cooperates with c-myc to immortalize pre-B cells
  Nature, 1988
• Ascorbate free radical reductase, a key enzyme of the ascorbic acid system
  FEBS Letters, 1981
• A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding
  Analytical Biochemistry, 1976
• Mechanism of free radical formation and disappearance during the ascorbic acid oxidase and peroxidase reactions
  Biochimica et Biophysica Acta, 1961