Cdk2 is dispensable for cell cycle inhibition and tumor suppression mediated by p27Kip1 and p21Cip1
- 1 June 2005
- journal article
- Published by Elsevier in Cancer Cell
- Vol. 7 (6) , 591-598
- https://doi.org/10.1016/j.ccr.2005.05.006
Abstract
No abstract availableKeywords
This publication has 36 references indexed in Scilit:
- Cdk2 Knockout Mice Are ViableCurrent Biology, 2003
- p27 as a target for cancer therapeuticsCancer Cell, 2003
- Deregulated degradation of the cdk inhibitor p27 and malignant transformationSeminars in Cancer Biology, 2002
- Inhibition of the Phosphoinositide 3-Kinase Pathway Induces a Senescence-like Arrest Mediated by p27Kip1Journal of Biological Chemistry, 2000
- The p21Cip1 and p27Kip1 CDK `inhibitors' are essential activators of cyclin D-dependent kinases in murine fibroblastsThe EMBO Journal, 1999
- p21 binding to PCNA causes G1 and G2 cell cycle arrest in p53-deficient cellsOncogene, 1998
- Differential Interaction of the Cyclin-dependent Kinase (Cdk) Inhibitor p27Kip1 with Cyclin A-Cdk2 and Cyclin D2-Cdk4Journal of Biological Chemistry, 1997
- Radiation-induced cell cycle arrest compromised by p21 deficiencyNature, 1995
- Mice Lacking p21CIP1/WAF1 undergo normal development, but are defective in G1 checkpoint controlCell, 1995
- Separate domains of p21 involved in the inhibition of Cdk kinase and PCNANature, 1995