Quantitation of amyloid-β peptides in biological milieu using a novel homogeneous time-resolved fluorescence (HTRF) assay
- 20 September 2000
- journal article
- Published by Elsevier in Journal of Neuroscience Methods
- Vol. 102 (1) , 61-68
- https://doi.org/10.1016/s0165-0270(00)00280-6
Abstract
No abstract availableKeywords
This publication has 28 references indexed in Scilit:
- Genetic dissection of Alzheimer's disease and related dementias: amyloid and its relationship to tauNature Neuroscience, 1998
- Alzheimer's Aβ(1–42) is generated in the endoplasmic reticulum/intermediate compartment of NT2N cellsNature Medicine, 1997
- Mutant presenilins of Alzheimer's disease increase production of 42-residue amyloid β-protein in both transfected cells and transgenic miceNature Medicine, 1997
- Evidence that the 42- and 40-amino acid forms of amyloid β protein are generated from the β-amyloid precursor protein by different protease activitiesProceedings of the National Academy of Sciences, 1996
- Familial Alzheimer's Disease–Linked Presenilin 1 Variants Elevate Aβ1–42/1–40 Ratio In Vitro and In VivoNeuron, 1996
- Increased amyloid-β42(43) in brains of mice expressing mutant presenilin 1Nature, 1996
- Amyloid β Protein (Aβ) in Alzheimeri's Disease BrainJournal of Biological Chemistry, 1995
- Mutation of the β-amyloid precursor protein in familial Alzheimer's disease increases β-protein productionNature, 1992
- Segregation of a missense mutation in the amyloid precursor protein gene with familial Alzheimer's diseaseNature, 1991
- Amyloid deposition as the central event in the aetiology of Alzheimer's diseaseTrends in Pharmacological Sciences, 1991