Endothelial nitric oxide synthase is strongly expressed in malignant mesothelioma but does not associate with vascular density or the expression of VEGF, FLK1 or FLT1
- 19 August 2001
- journal article
- research article
- Published by Wiley in Histopathology
- Vol. 39 (2) , 179-186
- https://doi.org/10.1046/j.1365-2559.2001.01211.x
Abstract
No abstract availableKeywords
This publication has 31 references indexed in Scilit:
- A Novel Function of VEGF Receptor-2 (KDR): Rapid Release of Nitric Oxide in Response to VEGF-A Stimulation in Endothelial CellsBiochemical and Biophysical Research Communications, 1999
- Vascular Endothelial Growth Factor Can Substitute for Macrophage Colony-Stimulating Factor in the Support of Osteoclastic Bone ResorptionThe Journal of Experimental Medicine, 1999
- VEGF-A Induces Expression of eNOS and iNOS in Endothelial Cells via VEGF Receptor-2 (KDR)Biochemical and Biophysical Research Communications, 1998
- Significant Correlation of Nitric Oxide Synthase Activity and p53 Gene Mutation in Stage I Lung AdenocarcinomaJapanese Journal of Cancer Research, 1998
- Nitric Oxide Synthase Activity in Human Lung CancerJapanese Journal of Cancer Research, 1997
- Production of vascular endothelial growth factor by human tumors inhibits the functional maturation of dendritic cellsNature Medicine, 1996
- Identification of the KDR tyrosine kinase as a receptor for vascular endothelial cell growth factorBiochemical and Biophysical Research Communications, 1992
- Molecular cloning and characterization of human endothelial nitric oxide synthaseFEBS Letters, 1992
- The fms -Like Tyrosine Kinase, a Receptor for Vascular Endothelial Growth FactorScience, 1992
- Malignant Pleural Mesothelioma: Some Aspects of Epidemiology, Differential Diagnosis and PrognosisPathology - Research and Practice, 1990