P53 function influences the effect of fractionated radiotherapy on glioblastoma tumors
- 1 January 1999
- journal article
- Published by Elsevier in International Journal of Radiation Oncology*Biology*Physics
- Vol. 43 (2) , 399-403
- https://doi.org/10.1016/s0360-3016(98)00393-9
Abstract
No abstract availableKeywords
This publication has 12 references indexed in Scilit:
- p53-Dependent G1 arrest and p53-independent apoptosis influence the radiobiologic response of glioblastomaInternational Journal of Radiation Oncology*Biology*Physics, 1996
- Accumulation of Nuclear p53 and Tumor Progression in Bladder CancerNew England Journal of Medicine, 1994
- p53-dependent apoptosis modulates the cytotoxicity of anticancer agentsCell, 1993
- Hematopoietic cells from mice deficient in wild-type p53 are more resistant to induction of apoptosis by some agentsBlood, 1993
- p53 mutations increase resistance to ionizing radiation.Proceedings of the National Academy of Sciences, 1993
- Thymocyte apoptosis induced by p53-dependent and independent pathwaysNature, 1993
- Multiple-fraction-per-day external beam radiotherapy for adults with supratentorial malignant gliomasJournal of Neuro-Oncology, 1992
- Identification of programmed cell death in situ via specific labeling of nuclear DNA fragmentation.The Journal of cell biology, 1992
- Radiation therapy for neoplasms of the brainJournal of Neurosurgery, 1987
- Intrinsic radiosensitivity of human cell lines is correlated with radioresponsiveness of human tumors: Analysis of 101 published survival curvesInternational Journal of Radiation Oncology*Biology*Physics, 1985