δ‐aminolevulinate dehydrase: a new genetic polymorphism in man

Abstract
A method has been developed for the electrophoretic and quantitative analyses of human red cell δ-aminolevulinate dehydrase (ALADH). The enzyme is under the control of an autosomal gene, with two common codominant alleles, ALADH1 and ALADH2, with frequencies of 0–89 and Oil, respectively, in the Italian population. Mean phenotypic enzyme activities are nearly identical: 52, 49 and 55 mlU/g Hb for ALADH 1, 2-1 and 2 phenotypes respectively.