Oncogenic Forms of NOTCH1 Lacking Either the Primary Binding Site for RBP-Jκ or Nuclear Localization Sequences Retain the Ability to Associate with RBP-Jκ and Activate Transcription
Open Access
- 1 April 1997
- journal article
- Published by Elsevier
- Vol. 272 (17) , 11336-11343
- https://doi.org/10.1074/jbc.272.17.11336
Abstract
No abstract availableKeywords
This publication has 36 references indexed in Scilit:
- Exclusive development of T cell neoplasms in mice transplanted with bone marrow expressing activated Notch alleles.The Journal of Experimental Medicine, 1996
- Physical interaction between a novel domain of the receptor Notch and the transcription factor RBP-Jκ/Su(H)Current Biology, 1995
- Suppressor of hairless directly activates transcription of enhancer of split complex genes in response to Notch receptor activity.Genes & Development, 1995
- The neurogenic suppressor of hairless DNA-binding protein mediates the transcriptional activation of the enhancer of split complex genes triggered by Notch signaling.Genes & Development, 1995
- Involvement of Notch-1 in mammalian retinal neurogenesis: association of Notch-1 activity with both immature and terminally differentiated cellsMechanisms of Development, 1995
- Signalling downstream of activated mammalian NotchNature, 1995
- Masking of the CBF1/RBPJ κ Transcriptional Repression Domain by Epstein-Barr Virus EBNA2Science, 1995
- Mediation of Epstein-Barr Virus EBNA2 Transactivation by Recombination Signal-Binding Protein J κScience, 1994
- TAN-1, the human homolog of the Drosophila Notch gene, is broken by chromosomal translocations in T lymphoblastic neoplasmsPublished by Elsevier ,1991
- A novel genetic system to detect protein–protein interactionsNature, 1989