Desensitization of prostacyclin responsiveness in platelets: Apparent differences in the mechanism in vitro or in vivo
- 15 August 1986
- journal article
- editorial
- Published by Elsevier in Biochemical Pharmacology
- Vol. 35 (16) , 2645-2649
- https://doi.org/10.1016/0006-2952(86)90169-3
Abstract
No abstract availableThis publication has 19 references indexed in Scilit:
- CLINICAL PHARMACOLOGY AND POTENTIAL OF PROSTACYCLINBritish Medical Bulletin, 1983
- DIVALENT CATIONS INCREASE [3H]‐PROSTACYCLIN BINDING TO MEMBRANES OF NEURONAL SOMATIC HYBRID CELLSBritish Journal of Pharmacology, 1981
- THE BINDING OF [3H]‐PROSTACYCLIN TO MEMBRANES OF A NEURONAL SOMATIC HYBRIDBritish Journal of Pharmacology, 1981
- Decreased sensitivity of human platelets to PGI2 during long-term intraarterial prostacyclin infusion in patients with peripheral vascular disease — A rebound phenomenon?Prostaglandins, 1981
- Selective Binding Site for [3H]Prostacyclin on PlateletsJournal of Clinical Investigation, 1979
- EFFECTS OF SULPHINPYRAZONE AND ASPIRIN ON PROSTAGLANDIN I2 (PROSTACYCLIN) SYNTHESIS BY ENDOTHELIAL CELLSBritish Journal of Pharmacology, 1978
- Differential formation of prostacyclin (PGX or PGI2) by layers of the arterial wall. An explanation for the anti-thrombotic properties of vascular endotheliumThrombosis Research, 1977
- Effects of prostacyclin (PGX) on cyclic AMP concentrations in human plateletsProstaglandins, 1977
- Modulation of human platelet adenylate cyclase by prostacyclin (PGX)Prostaglandins, 1977
- An enzyme isolated from arteries transforms prostaglandin endoperoxides to an unstable substance that inhibits platelet aggregationNature, 1976