On the Anti-inflammatory Mechanism of Hydrocortisone (Compound F)

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Abstract
Experimental studies indicate that the mechanism is referable to a suppression of cellular activity involving the injured cells at the site of inflammation, so that the chemical mediators, e.g., leukotoxine or the LPF (leukocytosis-promoting factor), are suppressed in their formation. These mediators in themselves constitute inflammation. By suppressing their formation, hydrocortisone becomes an anti-inflammatory agent.