REDUCED VASCULAR RESPONSE TO ANGIOTENSIN II IN SECONDARY HYPERALDOSTERONISM*

Abstract
The vascular sensitivity to intravenous angiotensin II has been compared in a group of 9 control subjects and in 14 patients with secondary hyperaldosteronism. Patients with secondary hyperaldosteronism had a diminished vasoconstrictor response to angiotensin snown by smaller rises in arterial blood pressure and in systemic vascular resistance. This was specific for angiotensin as 1-norepinephrine produced comparable changes in both groups.