Excess BAFF Rescues Self-Reactive B Cells from Peripheral Deletion and Allows Them to Enter Forbidden Follicular and Marginal Zone Niches
Top Cited Papers
- 1 June 2004
- Vol. 20 (6) , 785-798
- https://doi.org/10.1016/j.immuni.2004.05.010
Abstract
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This publication has 33 references indexed in Scilit:
- B Cell Receptor–independent Stimuli Trigger Immunoglobulin (Ig) Class Switch Recombination and Production of IgG Autoantibodies by Anergic Self-Reactive B CellsThe Journal of Experimental Medicine, 2003
- BAFF is a survival and maturation factor for mouse B cellsEuropean Journal of Immunology, 2002
- BAFF-R, a Newly Identified TNF Receptor That Specifically Interacts with BAFFScience, 2001
- An Essential Role for BAFF in the Normal Development of B Cells Through a BCMA-Independent PathwayScience, 2001
- TACI-Ig Neutralizes Molecules Critical for B Cell Development and Autoimmune DiseaseImmunity, 2001
- Marginal zone B cells exhibit unique activation, proliferative and immunoglobulin secretory responsesEuropean Journal of Immunology, 1997
- Outer Periarteriolar Lymphoid Sheath Arrest and Subsequent Differentiation of Both Naive and Tolerant Immunoglobulin Transgenic B Cells Is Determined by B Cell Receptor OccupancyThe Journal of Experimental Medicine, 1997
- Elimination of self-reactive B lymphocytes proceeds in two stages: Arrested development and cell deathCell, 1993
- Development and follicular localization of tolerant B lymphocytes in lysozyme/anti-lysozyme lgM/lgD transgenic miceInternational Immunology, 1992
- The Need for Central and Peripheral Tolerance in the B Cell RepertoireScience, 1990