Abnormal Bcl-2 and “Tissue” Transglutaminase Expression in Psoriatic Skin
- 1 December 1994
- journal article
- Published by Elsevier in Journal of Investigative Dermatology
- Vol. 103 (6) , 829-833
- https://doi.org/10.1111/1523-1747.ep12413590
Abstract
No abstract availableKeywords
This publication has 23 references indexed in Scilit:
- Apoptosis in human skin development: Morphogenesis, periderm, and stem cellsDevelopmental Dynamics, 1994
- Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programed cell deathCell, 1993
- Patterns of cell death: the significance of apoptosis for dermatologyExperimental Dermatology, 1993
- Identification of programmed cell death in situ via specific labeling of nuclear DNA fragmentation.The Journal of cell biology, 1992
- Induction of apoptosis in fibroblasts by c-myc proteinCell, 1992
- Mechanisms and Functions of Cell DeathAnnual Review of Cell Biology, 1991
- Interleukin-1 and Interleukin-6 in PsoriasisJournal of Investigative Dermatology, 1990
- Bcl-2 is an inner mitochondrial membrane protein that blocks programmed cell deathNature, 1990
- Apoptotic hepatocytes become insoluble in detergents and chaotropic agents as a result of transglutaminase actionFEBS Letters, 1989
- Psoriasis: a disease of abnormal Keratinocyte proliferation induced by T lymphocytesImmunology Today, 1986